Gibson, D. ., Harris, B. ., Prendergast, M. ., , JA, 2nd B., Holley, R. ., … Littleton, J. . (2003). Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor. Alcoholism, Clinical and Experimental Research, 27(7), 1099-106. https://doi.org/10.1097/01.ALC.0000075824.10502.DD

Mulholland, P. ., Harris, B. ., Wilkins, L. ., Self, R. ., Blanchard, J. ., Holley, R. ., … Prendergast, M. . (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.Y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)

Harris, B. ., Gibson, D. ., Prendergast, M. ., Blanchard, J. ., Holley, R. ., , … Littleton, J. . (2003). The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors. Alcoholism, Clinical and Experimental Research, 27(11), 1724-35. https://doi.org/10.1097/01.ALC.0000093601.33119.E3

Prendergast, M. ., Harris, B. ., Mullholland, P. ., JA, 2nd B., Gibson, D. ., Holley, R. ., & Littleton, J. . Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors. Neuroscience, 124(4), 869-77. https://doi.org/10.1016/j.neuroscience.2003.12.013 (Original work published 2004)

Mulholland, P. ., Self, R. ., Harris, B. ., Littleton, J. ., & Prendergast, M. . (-)-nicotine ameliorates corticosterone’s potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)

Butler, T. ., Self, R. ., Smith, K. ., Sharrett-Field, L. ., Berry, J. ., Littleton, J. ., … Prendergast, M. . (2010). Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N-methyl-D-asparate-type glutamate receptors. Neuroscience, 165(2), 525-34. https://doi.org/10.1016/j.neuroscience.2009.10.018 (Original work published 2010)

Mulholland, P. ., Self, R. ., Harris, B. ., Little, H. ., Littleton, J. ., & Prendergast, M. . (2005). Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures. Alcoholism, Clinical and Experimental Research, 29(5), 871-81. https://doi.org/10.1097/01.alc.0000163509.27577.da

Mulholland, P. ., Self, R. ., Stepanyan, T. ., Little, H. ., Littleton, J. ., & Prendergast, M. . Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro. Neuroscience, 135(4), 1129-39. https://doi.org/10.1016/j.neuroscience.2005.06.077 (Original work published 2005)

Mulholland, P. ., Stepanyan, T. ., Self, R. ., Hensley, A. ., Harris, B. ., Kowalski, A. ., … Prendergast, M. . Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience, 136(1), 259-67. https://doi.org/10.1016/j.neuroscience.2005.07.043 (Original work published 2005)

Mulholland, P. ., Self, R. ., Hensley, A. ., Little, H. ., Littleton, J. ., & Prendergast, M. . (2006). A 24 h corticosterone exposure exacerbates excitotoxic insult in rat hippocampal slice cultures independently of glucocorticoid receptor activation or protein synthesis. Brain Research, 1082(1), 165-72. https://doi.org/10.1016/j.brainres.2006.01.069 (Original work published 2006)